Apoplexy

Apoplexy

Pituitary kapa pituitary apoplexy ke lefu le sa tloaelehang empa le le kotsi. Ke tšohanyetso ea bongaka e hlokang taolo e nepahetseng.

Apoplexy ke eng?

Tlhaloso

Pituitary apoplexy ke lefu la pelo kapa tšollo ea mali le hlahang pituitary adenoma (hlahala e kotsi, e se nang mofets'e ea "endocrine" e hlahang mokokotlong oa pituitary bokong). Maemong a fetang halofo ea linyeoe, apoplexy e senola adenoma e sa faneng ka matšoao.

Lisosa 

Lisosa tsa pituitary apoplexy ha li utloisisoe ka botlalo. Pituitary adenomas ke lihlahala tse tsoang mali kapa tse shoang habonolo. Necrosis e ka bakoa ke khaello ea vascularization. 

Diagnostic

Litšoantšo tsa tšohanyetso (CT kapa MRI) li nolofalletsa ho etsa tlhahlobo ea lefu lena ka ho bontša adenoma nakong ea necrosis kapa hemorrhage. Mehlala ea mali e potlakileng le eona ea nkuoa. 

Batho ba amehang 

Pituitary apoplexy e ka hlaha ka nako efe kapa efe empa e atile haholo ho li-3 tsa hau. Banna ba amehile hanyane ho feta basali. Pituitary apoplexy e ama batho ba 2% ba nang le pituitary adenoma. Maemong a fetang 3 / XNUMX, bakuli ha ba hlokomele boteng ba adenoma ea bona pele ho bothata bo boima. 

Lintho tse kotsi 

Batho ba nang le lefu la pituitary adenoma hangata ba na le lintho tse ba hlaselang kapele kapa tse ba sitisang: ho noa lithethefatsi tse ling, litlhahlobo tse hlaselang, mafu a kotsi haholo (lefu la tsoekere, liteko tsa angiographic, mathata a coagulation, anti-coagulation, pituitary stimulation test, radiotherapy, bokhachane, kalafo ea bromocriptine, isorbide , chlorpromazine…)

Leha ho le joalo, lichapo tse ngata li etsahala ntle le tšitiso.

Matšoao a stroke

Pituitary kapa pituitary apoplexy ke motsoako oa matšoao a 'maloa, a ka hlahang ho feta lihora kapa matsatsi. 

Moriri oa hlooho 

Ho tšoaroa ke hlooho e bohloko ke letšoao la pele. Ho tšoaroa ke hlooho e pherese ho na le linyeoe tse fetang tse tharo. Li ka amahanngoa le ho nyekeloa ke pelo, ho hlatsa, feberu, pherekano ea tsebo, ka hona ho fihlela lefu la meningeal. 

Litšitiso tsa pono 

Maemong a fetang halofo ea pituitary apoplexy, pherekano ea pono e amahanngoa le hlooho. Tsena ke liphetoho tsa tšimo ea pono kapa tahlehelo ea matla a pono. E tloaelehileng ka ho fetisisa ke bitemporal hemianopia (tahlehelo ea tšimo ea pono ea mahlakore ka mahlakoreng a fapaneng a lebala la pono). Ho shoa litho tsa Oculomotor le hona ho atile. 

Matšoao a Endocrine 

Pituitary apoplexy hangata e tsamaisana le ho se sebetse hantle ha pituitary (hypopituitarism) e sa phetheheng ka mehla.

Kalafo bakeng sa pituitary apoplexy

Tsamaiso ea pituitary apoplexy ke mefuta e mengata ea lithuto tse fapaneng: lingaka tsa mahlo, lingaka tsa methapo ea mafu, lingaka tsa methapo ea kutlo le li-endocrinologists. 

Kalafo ea apoplexy hangata ke ea bongaka. Phetoho ea Hormonal e sebelisoa ho lokisa khaello ea endocrinological: kalafo ea corticosteroid, kalafo ea lihormone tsa qoqotho. Ho tsosolosoa ha hydro-electrolytic. 

Apoplexy e ka ba taba ea kalafo ea methapo ea pelo. Sena se ikemiselitse ho nyahamisa meaho ea lehae mme haholo-holo litselana tsa mahlo. 

Phekolo ea Corticosteroid e hlophisehile, ebang aoplexy e phekoloa ka methapo ea pelo kapa e behiloe leihlo ntle le ho etsoa opereishene (haholo-holo ho batho ba se nang tšimo ea pono kapa mathata a pono ea mahlo le ba sa holofalang). 

Ha ts'ebetso e potlakile, ho ka fola ka botlalo, ha ho ka etsahala hore kalafo e liehe ho ka ba le bofofu bo sa feleng kapa hemianopia. 

Likhoeling tse latelang tsa apoplexy, ho hlahlojoa bocha ha ts'ebetso ea pituitary ho etsoa ho bona hore na ho na le likhaello tse sa feleng tsa pituitary.

Thibela apoplexy

Ha ho hlile ha ho khonehe ho thibela li-pituitary apoplexies. Leha ho le joalo, ha ua lokela ho hlokomoloha matšoao a ka bang a pituitary adenoma, haholoholo pherekano ea pono (adenomas e ka hatella methapo ea mahlo). 

Ho buoa ha adenoma ho thibela karolo e 'ngoe ea pituitary apoplexy. (1)

(1) Arafah BM, Taylor HC, Salazar R., Saadi H., Selman WR Apoplexy ea pituitary adenoma kamora liteko tse matla tsa hormone ea gonadotropin Ke J Med 1989; 87: 103-105

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